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Case Based Learning for September 2025

A 68-year-old female presented to the ED with chest pain and shortness of breath. Her medical history was significant for hypertension, diabetes, and ESRD on dialysis for one year. She has been compliant with dialysis. Workup in the ED was significant for a junctional bradycardia in the 30s and a systolic blood pressure in the 60s. Lab work was significant for a glucose of 194, a potassium of 5.8, and a creatinine of 8.04 with a calculated GFR of 5. She was given 1mg atropine, which was ineffective. She was then started on an epinephrine infusion with minimal response. Transcutaneous pacing was initiated, and she was given pain control and sedation as her blood pressure tolerated. A tertiary care center was contacted and after an extended wait she was transferred. At the receiving hospital she was taken to the cath lab. No significant disease was noted, and a temporary venous pacemaker was placed. As her hyperkalemia was corrected and her Carvedilol wore off the pacemaker was discontinued. She was discharged home in good condition two days later. BRASH syndrome is an acronym for Bradycardia, Renal Failure, AV blockade, Shock, and Hyperkalemia. BRASH syndrome is a synergistic process created by a combination of hyperkalemia and medications blocking the atrioventricular (AV) node. The most common precipitants are hypovolemia and medications promoting hyperkalemia or renal injury. Often referred to as a vicious cycle, standard ACLS treatments may prove ineffective, as in this case. Bradycardia reduces the cardiac output; impairs renal perfusion; precipitates renal dysfunction; and exacerbates hyperkalemia. Left unchecked, the vicious cycle of hyperkalemia, bradycardia, renal dysfunction, and worsening hyperkalemia may progress to multiorgan failure with shock, bradycardia, and renal failure. The most common error in managing BRASH syndrome is fixating on a single component of the syndrome (e.g., hyperkalemia) and focusing solely…

Case Based Learning for August 2025

46-year-old male arrived to the ED in January via EMS who reported the patient had abdominal pain and shortness of breath and was initially anxious but then fell asleep en route.  On arrival, the patient was calm and kept his eyes closed without speaking much.  He mumbled that he was having trouble breathing recently and gave vague history of recent chest pain, abdominal pain, and coughing.  The patient fell asleep during history taking but was arousable to voice.  He had been seen the day before in our ED complaining of shortness of breath.   That chart revealed he had a history of methamphetamine abuse, cardiomyopathy, LV mural thrombus on Eliquis, and PTSD.  The day before, a full cardiac workup was unremarkable and he was discharged. The patient appeared withdrawn and somnolent, not speaking much.  I considered whether he was coming off of a meth binge, and experiencing the hypersomnia and encephalopathy sometimes seen with a meth crash.  It was noted that his hands and feet were cold and cyanotic, but the medics stated that the house was very cold and perhaps cold exposure was causing vasoconstriction.  Initial blood pressure was 100/70, heart rate of 76, SaO2 97%.  Initial EKG is shown below. The workup was ordered and the patient was rechecked and condition was not improving.  Pulse ox was not showing good waveform.  The medical student called me when the patient became agitated and profusely diaphoretic.  Subsequently, the patient said “I’m going to die!”, laid back, and lost consciousness.  He continued breathing and had a very weak femoral pulse.  While trying to decide whether to begin CPR, I noted a flurry of dysrhythmias on the monitor.  There were several beats of bradycardia followed by a few multifocal PVCs followed by a very wide-complex tachycardia with giant T waves with a…

Case Based Learning for July 2025

An Unusual Cause of Urticaria ER Presentation:  A 45 y/o female arrived by EMS because she was awoken while sleeping with abdominal pain, n/v, scattered hives and dyspnea. She felt lightheaded when ambulating but there was no syncope. Pertinent negatives:  no fever, cough, chest pain, throat pain or swelling in throat. Denied possible allergen exposure. Past medical history: noncontributory. Medications: none recently. Allergies: None known Social: no tobacco, alcohol or recreational drugs use. Physical exam: Vitals: Afebrile, HR 120 and regular, BP 100/70, RR 20 and mildly labored, O2 sat 97% RA Gen: Anxious-appearing female in mild respiratory distress. Skin: Classic urticarial lesions on back, legs and arms. HEENT: Normal. Neck: No adenopathy, thyromegaly, tenderness or swelling. Lungs: Wheezing in all fields with prolonged expiratory phase, no rales. CV: RRR, mild tachycardia, no audible murmurs or rubs. Puerperal pulses present in all extremities, 3 over 4 in intensity. Abdomen: Normal BS, nontender without masses or HSM. Medical decision making: This healthy patient appears to be having an allergic reaction and will need to be observed especially in view of the dyspnea, light headedness at home and soft BP with tachycardia on arrival to the ER. She is awake and alert, has no lip or tongue swelling, but angioedema is in the differential. Working diagnosis: allergic reaction, cause unknown. Differential diagnosis: the cause of hives is broad, hives with wheezing is usually caused by an exposure to an allergen, but other entities or processes must be considered. Such as: Scombroid: Caused by consuming fish containing scombroid (histamine fish poisoning) such as tuna, mackerel, mahi-mahi, and blue fish that are not properly refrigerated. Bacteria in these fish can produce high levels of histamine and thus as allergic type reaction. Serum Sickness: A type 3 (immune complex) hypersensitivity, seen in conditions such as…

Case Based Learning for June 2025

A 53-year-old female with past medical history of type 1 diabetes, asthma, anxiety, and depression, was brought in by EMS with CPR in progress. She called EMS with complaint of difficulty breathing. When EMS arrived, patient was talking and using a nebulizer treatment. Shortly after their arrival, she became unresponsive, apneic, and pulseless. CPR was initiated. An iGel was placed and Lucas chest compression device was placed. On rhythm check, no shock was advised. On arrival to the hospital, SpO2 was 55%, despite bilateral breath sounds and 100% oxygen and adequate bagging. An IV was placed and patient was given Narcan 2 mg, SoluMedrol 125 mg, and magnesium sulfate 2 g IV. She also received epinephrine 0.3 mg IM and a DuoNeb inline.  On rhythm check, sinus tachycardia with a pulse was noted and compressions discontinued. Patient continued to have only agonal respirations. The iGel was removed and she was intubated using etomidate and rocuronium. Her heart rate and blood pressure were increasing and a second dose of etomidate was given while a ketamine infusion was being initiated. EKG showed sinus tachycardia with rate 121 and RBBB. There was also ST depression in the inferior leads and inverted T waves in the lateral leads.  CXR showed ETT in good position 3 cm above the carina and no infiltrates or effusions. Labs: WBC 15.6, H/H 14.2/45.0, plt 322.  INR 1.0, PTT 33.0, dimer 812 (normal 100-599). VBG pH 6.786, pCO2 104, pO2 47, HCO3 15.8, O2 sat 41%. K 5.6, anion gap 23.6. Glucose 399. Trop-I (high sensitivity) 153. After receiving the VBG results, the patient was given bicarb 1 amp. Increased ventilatory rate was used to decrease ETCO2 down to the 40s. Accepting intensivist requested CT head and CTA chest/abd/pel and recommended 2 more amps of bicarb and ceftriaxone be…

Case Based Learning for May 2025

Following is the presentation a patient I recently had in the ED, where I felt I could have done better, especially with documentation. I think it brings up a number of good issues for possible learning/discussion, several of which I’ll mention below. I welcome any thoughts people have. – Gordan Luther, M.D.  HPI: 70 y.o. male, presented for evaluation of a possible stroke. He stated that the prior evening at about 8:00, almost 17 hours prior to his presentation, he had sudden onset of weakness of his left arm and leg. The weakness was severe enough that he had a hard time walking and he feels he had a couple light falls that did not result in injury. He denies any speech or visual change. The same areas have decreased sensation. He had no other complaints. He denied any palpitations, chest pain or SOB. He stated he did not have a history of diabetes despite the blood sugar in the 400s we found on arrival. There was almost no history in the EMR, and he stated he hadn’t seen a doctor for many years. PMH: None other than minor orthopedic injuries No allergies or meds. Lives alone. A friend talked him into coming in for evaluation. VS: BP 202/116 Pulse 102 Temp 36.7 °C Resp 20 SpO2 96% Physical Examination. General: No distress. Cooperative. Appears pale, although nontoxic. Head: Normocephalic. Atraumatic. Eyes: EOMI. PERRL. ENT: No nasal congestion. No pharyngeal erythema or exudate. Mucous membrane moist. Neck: Nontender. Supple. Lungs: Clear bilaterally. No rales, rhonchi, or wheezing. Heart: Regular rate and rhythm. Abdomen: Bowel sounds normal. Soft. Nontender. No rebound or guarding. No palpable masses. Neuro: Alert. Oriented x3. Speech fluent. Cranial nerves 2-12 intact. He has 4 out of 5 muscle strength on the left compared with 5 out…

ERX Community – Making a Difference in Bolivia

  Dr.Gordan Luther, ERx Site Director, recently spent several weeks in Bolivia where he continued a project with Medical Educators for Latin America (MELA). MELA provides trauma courses to Bolivian family doctors and firefighter paramedics. These professionals often handle trauma situations in the field that would typically be managed by paramedics in other countries, so the training is vital. Mano a Mano Bolivia organizes these courses, which have been incredibly well-received, with 50-60 students attending each year. Luther’s work with MELA began after he and his family spent a year volunteering in Cochabamba, Bolivia in 2009. While there, he connected with an NGO called Mano a Mano Bolivia. This NGO builds and maintains clinics and schools in rural areas of Bolivia. They have established over 170 clinics and 60 schools in the last 25 years, all of which remain operational and fully staffed. Inspired by their work, Luther and his family sought to support them upon returning home from that 2009/2010 volunteering year. Luther started taking groups down to Bolivia in about 2015. MELA is now a group of dedicated individuals, roughly half are medical doctors, and the other half firefighter paramedics. To support MELA efforts, the group holds an annual golf tournament fundraiser, and many members contribute personally. The initial goal was to fund one clinic, and now MELA is on their fifth project.  

Behind the Stethoscope: The Heart of ERx

In 2025, we are highlighting the medical directors of our ERx team. Their commitment to rural health care is the driving force behind our organization Juan Gabriel Velázquez D.O. Medical School Graduation: 2011 Joined ERx: 2024 Current position: ER Director of Bacon County Hospital What inspired you to pursue a career in medicine? “I grew up in a household of doctors: my grandfather, my father, and also my mother’s family. We would go for medical mission trips for vacation. Seeing the impact my parents had on people during those trips, I just fell in love with medicine at a very young age. I even tried to rebel in college, but I always came back to medicine. I have been in love with it ever since Why did you choose to work in a rural ER? My grandfather had a coffee plantation and cattle ranches. I was often at the farm helping out and being in the countryside. In medical school, every aspect of medicine was enjoyable. With every rotation, I had a feeling that it was my favorite.Eventually, it got to a point where I realized that I liked it all. My father being an ophthalmologist, he kind of wanted me to go into ophthalmology. One day, I told him that I wanted to be a real doctor. It broke his heart a little bit and I decided to go into family medicine. At first, I was at the bigger hospitals in Michigan. Then I wanted to move a little bit closer to my family so I moved to Georgia. The further I went, the smaller the hospitals got, the more I enjoyed medicine. I always had a passion for emergency medicine ever since my rotations in medical school. As a physician you see results quicker and with my background…

Case Based Learning for April 2025

Shoulder dislocation HPI: 20 yo otherwise healthy individual presents to you with acute left upper arm pain. The patient reports that they were mountain-bike riding, lost control of their bike and crashed. They are not sure how they landed but report immediate pain and numbness in their left upper extremity post-crash. Patient is concerned since they cannot move their upper arm and report it feels numb over the lateral aspect of their shoulder. They were able to ambulate into your emergency department and deny other injuries. They deny prior UE trauma, chronic joint pains, illnesses, or systemic symptoms.   Question 1: Based on your knowledge of anatomy, describe some potential causes of traumatic upper arm pain. Common causes of acute traumatic upper arm pain include humeral fracture, clavicular fracture, Acromioclavicular (AC) joint separation, glenohumeral dislocation, rotator cuff tear.   Question 2: What is the typical mechanism of injury for each of the above potential items? Think about the anatomy and how it might be disrupted. Proximal Humeral Fracture: Most often are caused by a fall or direct blow to the lateral shoulder. Type of fracture is determined by the body and arm position when the direct force is applied. (i.e., outstretched vs adducted arm) Clavicular Fracture: Often occurs with a fall onto a shoulder or being stuck over the clavicle with a heavy object. AC separation: Typically occurs from direct trauma to the superior or lateral aspect of the shoulder with the arm adducted Glenohumeral dislocation: An anterior shoulder dislocation is usually caused by a blow to the abducted, externally rotated, and extended arm. Examples would be fall on an outstretched arm, blocking a basketball shot, or blow to the posterior humerus. Rotator cuff tear: Most rotator cuff tears are seen in the middle aged or older adults and are…

Our ERx Communities – Tonasket, WA

The Okanogan River is a tributary of the Columbia River and runs about 115 miles long starting in southern British Columbia. Along its banks is Tonasket, Washington, which is one of ERx Clinical Partners communities. We work closely with the local hospital to provide emergency and hospitalist medical care. Our partnership with North Valley Hospital is over a decade old. In that time, we have built a partnership that has secured community trust in the Emergency Department and in acute care at NVH. Currently we are looking for an emergency medicine physician and an APP experienced in inpatient care to join our team. If you are considering a move to rural medicine, we would like to invite you to take a look at this beautiful area. The Okanogan River being one of the many reasons to consider this area. It is well-noted as a great place to fish for small-mouth bass and walleye.The river is also a habitat for diverse wildlife. This is all to say that if outdoor spaces are your answer to self-care, ERx has the place for you. Here are more details about the positions: ER physicians; https://zurl.to/Z57D?source=CareerSite APP Hospitalists: https://zurl.to/fqYg?source=CareerSite More about the area Okanogan County offers everything outdoor lovers want — rugged mountain peaks, high adrenaline adventure vacations, rural charm, endless outdoor adventure, wineries, concerts and fine dining! All this with the cost of living running 46% less than Seattle and a third less than Denver. Highlights: Low cost of living Wonderful small town with great farmers market 300 km of groomed Nordic ski trails More than 600 miles of trails suitable for snowmobiling Downhill skiing Mountain biking trails Whitewater Trail rides and rodeos Fishing and boating on the Columbia River, Okanogan River, and Beaner Lake. For more information about the Okanogan County Area of…

Case Report: Acute Angle-Closure Glaucoma in a 43-Year-Old Male

Introduction Acute angle-closure glaucoma (AACG) is an ocular emergency resulting from a rapid increase in intraocular pressure (IOP) due to obstructed aqueous humor outflow. It is commonly associated with a shallow anterior chamber angle, predisposing individuals to blockage. Without timely intervention, AACG can lead to permanent vision loss or blindness. This report describes the presentation, diagnosis, and management of AACG in a patient presenting to the emergency room (ER). Patient Presentation A 43-year-old male with no significant past medical history presented to the ER on a Sunday afternoon with blurred vision and pain in the right eye that began the day before. He initially hoped the symptoms would resolve on their own but sought medical attention after consulting a healthcare provider. He reported a sharp, constant headache behind the right eye rated 6/10, accompanied by nausea and blurred vision, described as though a film was over the right eye. He also noticed mild redness in the right eye but denied discharge, pain with eye movement, photophobia, fever, chills, respiratory, gastrointestinal, or genitourinary symptoms. Physical Examination Vital Signs: T 98.7°F, P 85 bpm, R 16/min, BP 151/87 mmHg, SpO₂ 97% on room air.Visual Acuity: Right Eye 20/40, Left Eye 20/20. Extraocular Movements (EOM): Full range of motion without pain. External Eye Exam: Eyelids everted—no foreign bodies. Mild conjunctival erythema, no discharge. Corneal Staining: Fluorescein stain showed no corneal uptake. Funduscopic Exam: Right Eye: Cloudy cornea, retina not visualized. Left Eye: Retina visualized, no gross abnormalities. Intraocular Pressure (IOP): Right Eye 76-80 mmHg, Left Eye 14-17 mmHg. Slit Lamp Exam: Not available in the ER. Working Diagnosis Acute Angle-Closure Glaucoma (AACG) Treatment Provided in ER Acetazolamide (Diamox): 500 mg PO (IV formulation unavailable) Timolol 0.5%: 2 drops in the right eye Apraclonidine and Prednisolone ophthalmic solutions were not available. Ophthalmology Consultation: Arranged…

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